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Abstract The increased environmental awareness coupled with the recent changes in the oil prices triggered the necessity of focusing on effective management of energy systems. Global climate change has caused many people to consider ways of reducing greenhouse gases Renewable energy has become an essential feature in curtailing emission of Green House Gases, while meeting the demand for energy. This paper presents an innovation system framework for development and diffusion of renewable energy technologies. The framework is used to identify opportunities for small and medium enterprises in the renewable energy sector. A case study on a successful development, installation and implementation of solar thermal systems households in Calgary, Alberta, by an entrepreneurial firm, is also presented.

Detailed analysis of ground rods and their influence on horizontal ground conductors, such as those forming grounding grids, is performed assuming a two layer soil stratification. The study starts with a discussion about the adequacy of uniform and two-layer soils as equivalent models for actual soil structures. Following this, a typical ground rod is analysed, while it is progressively associated with other ground rods, and ultimately, with horizontal conductors. The same procedure is also applied to an horizontal conductor. The results, shown using numerous charts which can be used conveniently for practical design purposes, lead to several interesting conclusions, many of which are new or still unpublished.

Abstract A transient three-dimensional model of a porous regenerator operating at room temperature is presented. The solid magnetic material and the regeneration fluid are modeled separately. The fluid flows through interstitial channels formed by a regular matrix of solid particles. The velocity, pressure and temperature fields are obtained from the simultaneous solution of the Navier–Stokes and energy equations with variable properties in the solid and the Boussinesq approximation for the fluid. The magnetocaloric effect (MCE) is taken into account by the inclusion of a source term in the energy equation for the magnetic solid. Special numerical schemes are used to avoid unrealistic computation time and memory requirements. Typical velocity fields and transient temperature profiles are presented.

Abstract The load-frequency control (LFC) problem is investigated on assumptions considered realistic for most power systems. Voltage and frequency dependence of system loads, and load inertia effects, are taken into account. Results include system dynamic response to changes in load, and effects of LFC parameter settings. Using a performance index based on squared tie-line flow error plus squared area frequency error, the effect of load representation on optimum integrator gain is examined.

This study was designed to test the hypothesis that chronic prenatal ethanol exposure decreases basal and stimulated L-glutamate release in the hippocampus of young, postnatal guinea pigs. Timed, pregnant guinea pigs were randomly assigned to one of the following three chronic treatment groups: 4 g ethanol/kg maternal body weight/day, isocaloric-sucrose and pair-feeding to the ethanol group, and water. Each oral treatment was given daily throughout gestation. Spontaneous locomotor activity was increased on postnatal day (PD) 10, and brain and hippocampal weights were decreased on PD 12 in the offspring of the ethanol group compared with the isocaloric-sucrose/pair-fed and water groups. On PD 12, the 45 mM K(+)- and 10 microM veratridine-stimulated release of glutamate in transverse hippocampal slices was decreased in the ethanol group compared with the two control groups. This alteration in glutamate release produced by chronic prenatal ethanol exposure may decrease the efficiency of excitatory synaptic transmission in the hippocampus during postnatal life.

Abstract Homogeneous charge compression ignition (HCCI) combustion is a spontaneous multi-site auto-ignition of a lean premixed fuel–air mixture, which has high heat release rate, short combustion duration and no evidence of flame propagation. In HCCI engines, there is no direct control method for the time of auto-ignition. Auto-ignition timing should be controlled in order to make heat release process take place at the appropriate time in the engine cycle. Heat release analysis is a diagnostic tool which aids engine experimenters. It facilitates the endeavors being conducted in obtaining a control method by investigating heat release rate and also cumulative heat release. This study can be divided into two parts. First, traditional first law heat release model which is widely used in engine combustion analysis was presented and the applicability of this model in HCCI engines was investigated. Second, a new heat release model based on the first law of thermodynamics accompanying with a temperature solver was developed and assessed. The model was applied in four test conditions with different operating conditions and a variety of fuel compositions, including i -octane, n -heptane, pure NG, and at last, a dual fueled case of NG and n -heptane. Results of this work indicate that utilizing the modified first law heat release model together with a solver for temperature correction will guarantee obtaining a well-behaved and accurate apparent heat release trend and magnitude in HCCI combustion engines.

AbstractActivation of potassium (K+) currents plays a critical role in the control of programmed cell death. Because pituitary adenylate cyclase‐activating polypeptide (PACAP) has been shown to inhibit the apoptotic cascade in the cerebellar cortex during development, we have investigated the effect of PACAP on K+ currents in cultured cerebellar granule cells using the patch‐clamp technique in the whole‐cell configuration. Two types of outward K+ currents, a transient K+ current (IA) and a delayed rectifier K+ current (IK) were characterized using two different voltage protocols and specific inhibitors of K+ channels. Application of PACAP induced a reversible reduction of the IK amplitude, but did not affect IA, while the PACAP‐related peptide vasoactive intestinal polypeptide had no effect on either types of K+ currents. Repeated applications of PACAP induced gradual attenuation of the electrophysiological response. In the presence of guanosine 5′‐[γthio]triphosphate (GTPγS), PACAP provoked a marked and irreversible IK depression, whereas cell dialysis with guanosine 5′‐[βthio]diphosphate GDPβS totally abolished the effect of PACAP. Pre‐treatment of the cells with pertussis toxin did not modify the effect of PACAP on IK. In contrast, cholera toxin suppressed the PACAP‐induced inhibition of IK. Exposure of granule cells to dibutyryl cyclic adenosine monophosphate (dbcAMP) mimicked the inhibitory effect of PACAP on IK. Addition of the specific protein kinase A inhibitor H89 in the patch pipette solution prevented the reduction of IK induced by both PACAP and dbcAMP. PACAP provoked a sustained increase of the resting membrane potential in cerebellar granule cells cultured either in high or low KCl‐containing medium, and this long‐term depolarizing effect of PACAP was mimicked by the IK specific blocker tetraethylammonium chloride (TEA). In addition, pre‐incubation of granule cells with TEA suppressed the effect of PACAP on resting membrane potential. TEA mimicked the neuroprotective effect of PACAP against ethanol‐induced apoptotic cell death, and the increase of caspase‐3 activity observed after exposure of granule cells to ethanol was also significantly inhibited by TEA. Taken together, the present results demonstrate that, in rat cerebellar granule cells, PACAP reduces the delayed outward rectifier K+ current by activating a type 1 PACAP (PAC1) receptor coupled to the adenylyl cyclase/protein kinase A pathway through a cholera toxin‐sensitive Gs protein. Our data also show that PACAP and TEA induce long‐term depolarization of the resting membrane potential, promote cell survival and inhibit caspase‐3 activity, suggesting that PACAP‐evoked inhibition of IK contributes to the anti‐apoptotic effect of the peptide on cerebellar granule cells.